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The Error Hypothesis of Mutation

Herbert E. Kubitschek
Science
New Series, Vol. 131, No. 3402 (Mar. 11, 1960), p. 730
Stable URL: http://www.jstor.org/stable/1705627
Page Count: 1
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Abstract

Accumulation of mutants in glucose-limited chemostats is proportional to growth rate, while in tryptophan-limited chemostats it is independent of growth rate. This behavior, which implies the failure of the error hypothesis, may be explainable on the basis of a unitary hypothesis: the results with glucose may be due to reversion or loss of latent mutants.

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